manbearpig
Don Juan
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- Apr 27, 2006
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If your game is tight enough, the muscles probably will not scare off the lot of them.
How the fu<k can you get by on 50g of protein a day? (and 10-15g on non workout days?) Sorry but I call bull****.MotoXXX said:I try to stay under 2,000 calories a day and at least 50g protein on my weights days and 10-15g on non weight days. Thats it really. I can get more in depth in the forum for fitness if anyone wants.
MotoXXX said:When I do approach though it is probably 90% successful.
he's nowhere near that lean I guarantee itAgonyUncle said:Look guys, the trick is not for him to change the way he looks, its for him to change the way he thinks. Jesus, at 240lbs and 8% BF, he is massive.
he competed at around 223 at 3% bodyfat meaning that he was around 240 at 8% at the time in the offseason and yes it is hard to believe because it doesn't happen naturallyAgonyUncle said:Er, try closer to 2-3% BF, which is the level they compete at. 8% is not that low a BF% for a bodybuilder.
I have a mate that is almost that size. Its not that hard to believe
actually no there is no proof that they have caused any medical problems in any human beingViktor said:Steroids are known to cause medical problems, impotence etc. Someone who may have been a steroid user is a poor bet for a mate.
Wrong. Look below for quotes from a recent review article on the damage they cause.Silverback82 said:actually no there is no proof that they have caused any medical problems in any human being
as for being a poor bet for a mate, maybe so but ever hear of bad boys?
obviously you didn't watch the video I postedViktor said:Wrong. Look below for quotes from a recent review article on the damage they cause.
Also, the widely-held perception among the public is that they DO cause damage, and part of that damage is reproductive system damage ie: testicular shrinkage, etc.
This is not sexy.
And pumping yourself up into a musclebound caricature is evidence of overcompensating for some psychological problem and also of obsession.
Also not sexy.
Here's information for you on problems caused by anabolic steroids:
Adverse effects of anabolic steroids in athletes: A constant threat
Toxicology Letters Volume 158, Issue 3 , 15 September 2005, Pages 167-175
Anabolic-androgenic steroids (AAS) are used as ergogenic aids by athletes and non-athletes to enhance performance by augmenting muscular development and strength. AAS administration is often associated with various adverse effects that are generally dose related. High and multi-doses of AAS used for athletic enhancement can lead to serious and irreversible organ damage. Among the most common adverse effects of AAS are some degree of reduced fertility and gynecomastia in males and masculinization in women and children. Other adverse effects include hypertension and atherosclerosis, blood clotting, jaundice, hepatic neoplasms and carcinoma, tendon damage, psychiatric and behavioral disorders. More specifically, this article reviews the reproductive, hepatic, cardiovascular, hematological, cerebrovascular, musculoskeletal, endocrine, renal, immunologic and psychologic effects.
3.1. Reproductive effects
Use of steroids in men decreases levels of luteinizing hormone and follicle-stimulating hormones, which leads to decreased endogenous testosterone production, decreased spermatogenesis, and testicular atrophy. The testicular atrophy and the oligospermia or azoospermia usually resolve after discontinuation of the drugs, but the count and morphology of the sperm may be abnormal for up to 6 months (Boyadjiev et al., 2000, Dohle et al., 2003 and Eklof et al., 2003). Prostatic hypertrophy, priapism, and, rarely, carcinoma of the prostate can be associated with steroid use (Wemyss-Holden et al., 1994). Gynecomastia may result from the peripheral conversion of androgens to estradiol and estrone.
3.2. Hepatic effects
Elevations in levels of liver enzymes (aspartate aminotransferase, alanine aminotransferase, and lactate dehydrogenase) are also common in athletes who use steroids. Hepatic dysfunction is most commonly associated with the 17-alpha alkylated steroids (Friedl, 2000 and Snyder, 2001). Cholestatic jaundice occurs occasionally with steroid use and typically resolves within 3 months of discontinuing the drugs. Liver tumors, both benign and malignant, have been linked to the administration of steroids (Watanabe and Kobayashi, 1993, Soe et al., 1994, Friedl, 2000 and Velazquez and Alter, 2004). Several athletes with extensive histories of steroid use have died of hepatocellular carcinoma or of hepatic tumor rupture.
3.3. Cardiovascular, cerebrovascular and hematological effects
A significant decrease in high-density lipoprotein (HDL) cholesterol and, often, an increase in low-density lipoprotein (LDL) cholesterol occur with steroid use, placing the user at increased risk for atherosclerotic heart disease. Cholesterol levels may return or not to normal following cessation of steroid use and normalization depends on the duration of AAS abuse (Cohen et al., 1988, Glazer, 1991, Shahidi, 2001 and Hartgens et al., 2004).
Hypertension is associated with anabolic steroid use and myocardial infarction has been reported in several athletes who used steroids for a prolonged period of time (Ferenchick, 1990). These case reports reveal that the actual frequency of myocardial infarction and sudden death among users of AAS is presumably underreported in medical literature and although a causal relationship has not yet been established, a pathogenic effect is plausible (Ferenchick and Adelman, 1992, Rockhold, 1993, Ansell et al., 1993 and Halvorsen et al., 2004). Moreover, anabolic use has been related to irreversible changes to myocardium, such as concentric left ventricular hypertrophy (Urhausen et al., 2004).
3.4. Musculoskeletal effects
Of particular concern is the premature epiphyseal closure in any child/adolescent, which results in a decrease in adult height after prolonged exposure to androgens (Al-Ismail et al., 2002). Some scientists believe that there is an increased risk of musculotendious injuries with steroid use. Tendons may not increase in strength as muscles do and, when subject to increased intensity and frequency of training, may be at higher risk for rupture.
3.6. Renal effects
Acute renal failure as a complication of rhabdomyolysis in a body builder using AAS has been reported (Hageloch et al., 1988). The combination of AAS and creatinine supplement that has been currently abused by body builders may cause renal damage. A case of diffuse membranoproliferative glomerulonephritis has been reported (Revai et al., 2003).
Wilm's tumor, uncommon in adults, has been reported in several athletes using anabolic steroids (Joyce, 1991). There is evidence suggesting that steroids are weak carcinogens that can initiate tumor growth or promote such growth in the presence of other carcinogens (Lamb, 1984 and Watanabe and Kobayashi, 1993).
3.8. Psychologic effects
Some individuals may experience mental status and behavioral changes with anabolic steroid use, including irritability, aggressiveness, euphoria, depression, mood swings, altered libido, and even psychosis (Kashkin and Kleber, 1989, Bahrke et al., 1990, Middleman and DuRant, 1996 and Clark and Henderson, 2003).
A recent study of health club athletes revealed that 90% of users reported episodes of over aggressiveness and violent behavior which were believed to be induced by steroids (Tamir et al., 2004), whereas other studies do not support any association between AAS and aggressive behavior (Bhasin et al., 1996a, Bhasin et al., 1996b and Yates et al., 1999).
Anabolic steroid withdrawal and dependency disorders have also been reported (Foley and Schydlower, 1993 and Bahrke, 2000). Acute anabolic steroid withdrawal may produce symptoms of central non-adrenergic hyperactivity including anxiety, irritability, insomnia, hot flashes, sweats, chills, anorexia, myalgia, nausea, vomiting, piloerection, tachycardia, and hypertension. Depression and anabolic steroid craving may also occur with withdrawal. Nevertheless, high risk psychological behaviors may also be the primary problem of those who take anabolic steroids although relative psychiatric disorders may be observed on these users prior to use (Middleman and DuRant, 1996).
Steroids may be psychologically addicting, even meeting the DSM-IV criteria for psychoactive substance dependence in some cases. Electroencephalogram changes similar to those seen with psycho-stimulant drugs have been reported with steroid use (Frankle et al., 1988). Physical withdrawal symptoms, similar to those seen in opiate withdrawal, have occurred upon cessation of extremely high doses of steroids. Moreover, the developing nervous system of children may be especially vulnerable to the psychological effects of steroids. Adolescents may lack the maturity to cope with possible drug-induced mood changes. In addition, the development of appropriate social skills and controls necessary to deal with pubertal changes may be made difficult if changes occur more rapidly than expected (Snyder, 2001).
There have also been reports stating that there is a relationship between hormone levels (gonadotropins, gonadal steroid hormones, and adrenal androgens) and the emotional dispositions and aggressive behaviors of adolescents. The results indicate that high hormone levels were related to potentially adverse psychological consequences for boys and girls (Clark and Henderson, 2003).