Herbs/Supps for anxiety relief...?
- Thread starter silverwex
- Start date
Don't have much time right now so I can't explain how they work and all but here's a list of a few more:
Passionflower
Valerian
Theanine (extracted from Green Tea)
Hops
Rhodiola
5-HTP (evidence is still out on this one to some extent)
Tyrosine (helps with Dopamine and Norepinephrine levels in the brain)
Passionflower
Valerian
Theanine (extracted from Green Tea)
Hops
Rhodiola
5-HTP (evidence is still out on this one to some extent)
Tyrosine (helps with Dopamine and Norepinephrine levels in the brain)
CapiCrimini
Master Don Juan
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Cannibas Puff
Magic Dragon
Mary Jane
Dro
Purple Haze
or any form of weed... BUT... I do know a few plants that will help...
Catnip
Black Willow
Wild Mint
Common St. JohnsWort[like you said]
MotherWort
Peppermint
Ladys Slipper
Hawthorn
Pitcher Plant
All of the above you'd need to find the wild plant and steep[the leaves or pines] it a pint of water for 10 minutes...
[Note: I only mentioned common plants so you should be able to find them... ONLY USE IF YOU ARE CERTIAN YOU HAVE IDENTIFIED THE RIGHT PLANT. You should learn all the look alike plants especially the poisioness ones. Also *cough**cough* One of the above list is a highly laxitive plant. If you take it you won't be having fun
. Thats just there so you'll be certian to look up the plants before taking them ]
As for the results I've taken them before... besides St. JohnsWort. I'm not sure if they come in pills or anything. Medicinal plants arn't my forte' I know Bug Repelants, and cures for some minor things like itching, diahrea, and colds and fever. Sides that I mainly know edibles...
Good luck. try searching
Magic Dragon
Mary Jane
Dro
Purple Haze
or any form of weed... BUT... I do know a few plants that will help...
Catnip
Black Willow
Wild Mint
Common St. JohnsWort[like you said]
MotherWort
Peppermint
Ladys Slipper
Hawthorn
Pitcher Plant
All of the above you'd need to find the wild plant and steep[the leaves or pines] it a pint of water for 10 minutes...
[Note: I only mentioned common plants so you should be able to find them... ONLY USE IF YOU ARE CERTIAN YOU HAVE IDENTIFIED THE RIGHT PLANT. You should learn all the look alike plants especially the poisioness ones. Also *cough**cough* One of the above list is a highly laxitive plant. If you take it you won't be having fun
. Thats just there so you'll be certian to look up the plants before taking them ]As for the results I've taken them before... besides St. JohnsWort. I'm not sure if they come in pills or anything. Medicinal plants arn't my forte' I know Bug Repelants, and cures for some minor things like itching, diahrea, and colds and fever. Sides that I mainly know edibles...
Good luck. try searching
CapiCrimini
Master Don Juan
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oh yeah...
To R3N's post... If you take pure Lavender extract... I belive it's
20 drops of Lavender oil to 6 oz of water and put it in a squirt bottle... If you spray it on your headboard it will help you sleep. expensie but it calms me down... stinks a liitle at first though... strong ****
To R3N's post... If you take pure Lavender extract... I belive it's
20 drops of Lavender oil to 6 oz of water and put it in a squirt bottle... If you spray it on your headboard it will help you sleep. expensie but it calms me down... stinks a liitle at first though... strong ****
CapiCrimini
Master Don Juan
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what do you mean? If you mix it yourself you know the quality...
I went to china and tasted real chinese tea, I was blown away. really good ****... then you come back and you taste the crappy **** that comes out of the box...
That and i spend a lot of time out in woods Making things naturally seems a lot better to me... though I don't study it extensively I do know a little about stuff like this.
The Bad Ass Canadian
Master Don Juan
Chamomile is an excellent tea to reduce anxiety.
wanna get rid of your anxiety, forever?
go here.http://www.panic-anxiety.com/ This guy has the only medication/therapy free way of getting rid of this nasty, life sucking disorder.
I went through the worst of my anxiety, this past year. It was utter hell. That sick / nervous feeling you get in the pit of your stomach, when something bad is gonna happen X100, all day everyday for a month straight.
It was awful.
I bought the program from this guy, and now my anxiety is gone, completely. No meds.
Don't screw around, thinking that herbal supplements will help, cause they won't.
The anxiety is all in your head, literally. It's a force of habit, that your concious mind has developped, and you need to break out of the cycle to get better.
The Bad Ass Canadian
wanna get rid of your anxiety, forever?
go here.http://www.panic-anxiety.com/ This guy has the only medication/therapy free way of getting rid of this nasty, life sucking disorder.
I went through the worst of my anxiety, this past year. It was utter hell. That sick / nervous feeling you get in the pit of your stomach, when something bad is gonna happen X100, all day everyday for a month straight.
It was awful.
I bought the program from this guy, and now my anxiety is gone, completely. No meds.
Don't screw around, thinking that herbal supplements will help, cause they won't.
The anxiety is all in your head, literally. It's a force of habit, that your concious mind has developped, and you need to break out of the cycle to get better.
The Bad Ass Canadian
Alpine
Master Don Juan
Hey Bad Ass, I've just gone through that link and I'm anxious that I'll buy it and it won't work.
nothing posted here is going to compare to piricetam, aniracetam, phenbuit. heres some studies on each one of the nootropics i mentioned:
Piracetam (2-oxo-1-pyrrolidine acetamide) is the most well-known nootropic agent. The word nootropic comes from Greek noos (mind) and tropein (towards), and refers to a class of drugs which have common properties including enhancement of learning and memory, enhancement of the resistance of the brain towards chemical and physical injuries, and a general lack of stimulant or sedative properties or other side effects. Piracetam was the first compound discovered to have all of these properties, and became the prototypical nootropic which serves as the reference drug for newer nootropics. Although the definition and requirements are a common topic of debate, a drug is generally considered a nootropic only if it improves memory in the absence of a cognitive deficit [7]. In clinical practice, nootropics are commonly used to treat brain distrubances caused by various chemical and physical agents, for the treatment of dementia and mild cognitive impairment, and as cerebroprotective agents in stroke [3].
Aniracetam (1-(p-anisoyl)-2 pyrrolidinone; 1-(4 methoxybenzoyl) 2-pyrrolidinone) is a cognitive enhancer related to piracetam, but approximately 5-10 times as potent. Clinical studies in the elderly have found it to have nootropic, anxiety-reducing, and antidepressant properties. Studies in many animal species, including monkeys, have found that aniracetam improves cognition in both healthy adult animals and animals impaired by a variety of drugs and conditions, and it improves cognitive performance on some tests in which piracetam is ineffective. Aniracetam functions through multiple mechanisms, including positive modulation of AMPA receptors and increasing acetylcholine, dopamine, and serotonin release in some areas of the brain. Like piracetam, it is virtually non-toxic and associated with very few side effects. It may cause insomnia in some individuals if taken close to bedtime. The effect after a single dose of aniracetam peaks two hours after administration and reaches baseline by the six hour point. Recommended dosage is 750-1500 mg daily, or lower if used in conjunction with other nootropics.
Phenibut (beta-phenyl- gamma-aminobutyric acid, also spelled fenibut, originally known as phenigamma) is a derivative of the neurotransmitter GABA that crosses the blood-brain barrier [1]. It was developed in Russia, and there it has been used clinically since the 1960's for a range of purposes. Phenibut has both nootropic and anxiolytic (anxiety-reducing) properties, and it is commonly compared to diazepam (Valium), baclofen, and piracetam, and it has similarities to and differences from all of these substances.
Piracetam (2-oxo-1-pyrrolidine acetamide) is the most well-known nootropic agent. The word nootropic comes from Greek noos (mind) and tropein (towards), and refers to a class of drugs which have common properties including enhancement of learning and memory, enhancement of the resistance of the brain towards chemical and physical injuries, and a general lack of stimulant or sedative properties or other side effects. Piracetam was the first compound discovered to have all of these properties, and became the prototypical nootropic which serves as the reference drug for newer nootropics. Although the definition and requirements are a common topic of debate, a drug is generally considered a nootropic only if it improves memory in the absence of a cognitive deficit [7]. In clinical practice, nootropics are commonly used to treat brain distrubances caused by various chemical and physical agents, for the treatment of dementia and mild cognitive impairment, and as cerebroprotective agents in stroke [3].
Aniracetam (1-(p-anisoyl)-2 pyrrolidinone; 1-(4 methoxybenzoyl) 2-pyrrolidinone) is a cognitive enhancer related to piracetam, but approximately 5-10 times as potent. Clinical studies in the elderly have found it to have nootropic, anxiety-reducing, and antidepressant properties. Studies in many animal species, including monkeys, have found that aniracetam improves cognition in both healthy adult animals and animals impaired by a variety of drugs and conditions, and it improves cognitive performance on some tests in which piracetam is ineffective. Aniracetam functions through multiple mechanisms, including positive modulation of AMPA receptors and increasing acetylcholine, dopamine, and serotonin release in some areas of the brain. Like piracetam, it is virtually non-toxic and associated with very few side effects. It may cause insomnia in some individuals if taken close to bedtime. The effect after a single dose of aniracetam peaks two hours after administration and reaches baseline by the six hour point. Recommended dosage is 750-1500 mg daily, or lower if used in conjunction with other nootropics.
Phenibut (beta-phenyl- gamma-aminobutyric acid, also spelled fenibut, originally known as phenigamma) is a derivative of the neurotransmitter GABA that crosses the blood-brain barrier [1]. It was developed in Russia, and there it has been used clinically since the 1960's for a range of purposes. Phenibut has both nootropic and anxiolytic (anxiety-reducing) properties, and it is commonly compared to diazepam (Valium), baclofen, and piracetam, and it has similarities to and differences from all of these substances.
Structurally, phenibut is similar to GABA, baclofen (p-Cl-phenibut), and beta-phenylethylamine (PEA). GABA is the primary inhibitory neurotransmitter in the brain. The addition of the phenyl ring to GABA allows the compound to more easily cross the blood-brain barrier, but also changes its activity profile [1-2]. Baclofen is a drug commonly used in studies on GABA(B) receptors, and also clinically used to treat severe spasticity of cerebral origin [3]. PEA is a naturally occuring biogenic amine which is similar in structure to amphetamine, and like amphetamine, it is a stimulant that causes the release of dopamine, and also promotes anxiety in high enough amounts.
Phenibut is a GABA receptor agonist and also causes the release of GABA. Similar to baclofen, phenibut is an agonist at GABA(B) receptors, although it does have some effect on GABA(A) receptors as well [2]. It is possible that phenibut has a higher activity at central GABA(B) receptors than peripheral ones [4]. The role of the GABA(B) receptor is not well-established, although research in the last seven years has significantly increased our understanding of this receptor. The most well-established role of GABA(B) receptors is inhibition of the release of some neurotransmitters, and it may also serve as a negative feedback mechanism for GABA release [5-6].
Because of the structural similarity to PEA, phenibut may share some similarities and differences with it. When phenibut is administered along with PEA, it antagonizes many of its effects, such as promotion of anxiety, promotion of seizures, and hyperthermia. This has lead some to postulate that antagonism of PEA, rather than the GABA-mimetic activity, may be the important mechanism of action for tha anxiolytic effect of phenibut [2, 7]. Phenibut also increases dopamine levels, and it has been postulated that the structural similarity to PEA may play a role in this effect [2].
There is one report in the literature of serotonergic effects of phenibut [8], but it does not look as though this has been followed up on.
Effects of phenibut
Anxiety reduction. Phenibut is effective in many animal models of anxiety, although there is often dependence on study conditions. In cats classified as "anxious" or "passive," phenibut reduced the fear response and increased aggression in a confrontational situation, while it had no effect on aggressive cats. In normal cats, it lead to "positive emotional symptoms" [2]. In mice, phenibut increased social behavior [9]. In rats, phenibut decreased some of the physiological responses to stress, including the elevation of glucocorticoid levels [10]. Phenibut has also been reported to decrease the fear response caused by electrical stimulation and counteract the anxiogenic effect of the beta-carboline DMCM [2, 11]. Studies in rats examined the behavioral properties of phenibut when it was administered locally into different parts of the brain, and it usually lead to a reduction of anxiety in one or more models [12-16].
The results of animal models don't always pan out in the real world, however, phenibut has a mechanism of action similar to that of many drugs which are known to reduce anxiety in humans. Animal studies have compared the profile of phenibut to diazepam (Valium), which has pronounced anxiolytic properties, and piracetam, which has weak anxiolytic properties. One study found phenibut had a tranquilizing effect similar to, but weaker than diazepam. It also caused sedation and muscle relaxation (whereas piracetam did not), but again these effects were weaker than those caused by diazepam [2].
In Russia, phenibut is commonly used to treat many neuroses, including post-traumic stress disorder, stuttering, and insomnia. In double blind placebo-controlled studies, phenibut has reportedly been found to improve intellectual function, improve physical strength, and reduce fatigue in neurotic and psychotic patients [2].
Nootropic effects. Although phenibut does not meet all the requirements of a nootropic, it does have many similarities to piracetam. In mice, phenibut causes significant improvement on the passive avoidance test [2]. In this test of memory, animals are put in an undesirable area (such as a lighting situation or height from the floor that that species dislikes), and then given a negative stimulus (such as a shock) when they exit that area. Their ability to stay in the original area reflects how well they remember that if they exit it, they will receive the undesirable stimulus. Phenibut also improves performance on the swimming and rotarod tests and antagonizes the amnestic effect of chloramphenicol [2]. It also has an antihypoxic effect, a trait commonly seen among nootropics [17]. However, in one study, phenibut was ineffective in the water maze and shuttle box tests, while piracetam was [18]. Other research supports the idea that phenibut has nootropic activity similar to that of piracetam, but not as strong [19]. Nootropic activity has also been reported in humans [2], but it was not specified whether these were healthy adult humans, and they were probably elderly or psychiatric patients.
Another trait phenibut shares with nootropics is neuroprotection. Multiple animal studies have indicated that phenibut administration increases resistance to the detrimental effects of edema on mitochondria and energy production in the brain [20-22]. Phenibut also normalizes brain energy metabolism changes caused by chronic stress [23]. It was found to prevent changes in plasma electrolytes caused by cerebral injury [24]. Phenibut also protects dopaminergic neurons, and improved the condition of patients being treated with antiparkinsonic drugs [25].
Other effects. Phenibut has anticonvulsant activity against some drugs or conditions, but not others. It also potentiates the action of some other anticonvulsant drugs, and has been used to treat patients with epilepsy [2]. Phenibut has been reported to reduce motion sickness, and used in the treatment of alcohol and morphine withdrawal [2, 26]. One study indicated that phenibut increased resistance to heat stress and improved working capacity in humans [27].
Some studies indicate that phenibut has anti-arrhythmic properties in humans [28-29]. It also has other cardioprotective properties [30-31]. Finally, phenibut showed promise in experimental models of gastric lesions [32-33].
Side effects and suggested use
Phenibut has low acute toxicity. Reported LD50s (dose required to kill 50% of laboratory animals) are 900 mg/kg i.p. in mice, 700 mg/kg i.p. in rats, and 1000 mg/kg in rats (method of administration not given) [2, 34]. Chronic administration of 50 mg/kg did not have teratogenic effects in rats [34]. In clinical studies, no signs of toxicity have been reported, and side effects are few. Some report drowsiness, but this effect is not nearly as likely or severe as with benzodiazepines [2].
One should be aware of the potential for drug interactions when taking phenibut. In many cases, it will decrease the threshold dose and potentiate certain actions of a drug. It amplifies some of the effects of anesthetics (ether, chloral hydrate, and barbiturates), diazepam, alcohol, and morphine [2, 35-36]; it would also presumably have an interaction with related drugs, such as other opiates and GHB. In contrast, taking phenibut with some other drugs, such as stimulants, will more than likely just blunt their effect.
In humans, the plasma half-life after a 250 mg oral dose of phenibut is 5.3 hours, and most of the administered drug is excreted unchanged [2]. Reported dosages used in clinical studies range from 250 to 1500 mg daily, usually divided among three doses [2, 37]. Feedback indicates that the ideal dose may be in the higher end of this range.
Tolerance develops to many of the effects of phenibut, although it is reported that it does not develop to the nootropic effect. The first signs of tolerance may be seen within as little as five days. For this reason, it is commonly used for one to two week periods, or dosage is increased by 25-30% after two weeks [2]. This makes phenibut ideal for short periods of stress or anxiety, but not ideal for chronic use. It is possible that taking only one dose daily may partially reduce the development of tolerance
Phenibut is a GABA receptor agonist and also causes the release of GABA. Similar to baclofen, phenibut is an agonist at GABA(B) receptors, although it does have some effect on GABA(A) receptors as well [2]. It is possible that phenibut has a higher activity at central GABA(B) receptors than peripheral ones [4]. The role of the GABA(B) receptor is not well-established, although research in the last seven years has significantly increased our understanding of this receptor. The most well-established role of GABA(B) receptors is inhibition of the release of some neurotransmitters, and it may also serve as a negative feedback mechanism for GABA release [5-6].
Because of the structural similarity to PEA, phenibut may share some similarities and differences with it. When phenibut is administered along with PEA, it antagonizes many of its effects, such as promotion of anxiety, promotion of seizures, and hyperthermia. This has lead some to postulate that antagonism of PEA, rather than the GABA-mimetic activity, may be the important mechanism of action for tha anxiolytic effect of phenibut [2, 7]. Phenibut also increases dopamine levels, and it has been postulated that the structural similarity to PEA may play a role in this effect [2].
There is one report in the literature of serotonergic effects of phenibut [8], but it does not look as though this has been followed up on.
Effects of phenibut
Anxiety reduction. Phenibut is effective in many animal models of anxiety, although there is often dependence on study conditions. In cats classified as "anxious" or "passive," phenibut reduced the fear response and increased aggression in a confrontational situation, while it had no effect on aggressive cats. In normal cats, it lead to "positive emotional symptoms" [2]. In mice, phenibut increased social behavior [9]. In rats, phenibut decreased some of the physiological responses to stress, including the elevation of glucocorticoid levels [10]. Phenibut has also been reported to decrease the fear response caused by electrical stimulation and counteract the anxiogenic effect of the beta-carboline DMCM [2, 11]. Studies in rats examined the behavioral properties of phenibut when it was administered locally into different parts of the brain, and it usually lead to a reduction of anxiety in one or more models [12-16].
The results of animal models don't always pan out in the real world, however, phenibut has a mechanism of action similar to that of many drugs which are known to reduce anxiety in humans. Animal studies have compared the profile of phenibut to diazepam (Valium), which has pronounced anxiolytic properties, and piracetam, which has weak anxiolytic properties. One study found phenibut had a tranquilizing effect similar to, but weaker than diazepam. It also caused sedation and muscle relaxation (whereas piracetam did not), but again these effects were weaker than those caused by diazepam [2].
In Russia, phenibut is commonly used to treat many neuroses, including post-traumic stress disorder, stuttering, and insomnia. In double blind placebo-controlled studies, phenibut has reportedly been found to improve intellectual function, improve physical strength, and reduce fatigue in neurotic and psychotic patients [2].
Nootropic effects. Although phenibut does not meet all the requirements of a nootropic, it does have many similarities to piracetam. In mice, phenibut causes significant improvement on the passive avoidance test [2]. In this test of memory, animals are put in an undesirable area (such as a lighting situation or height from the floor that that species dislikes), and then given a negative stimulus (such as a shock) when they exit that area. Their ability to stay in the original area reflects how well they remember that if they exit it, they will receive the undesirable stimulus. Phenibut also improves performance on the swimming and rotarod tests and antagonizes the amnestic effect of chloramphenicol [2]. It also has an antihypoxic effect, a trait commonly seen among nootropics [17]. However, in one study, phenibut was ineffective in the water maze and shuttle box tests, while piracetam was [18]. Other research supports the idea that phenibut has nootropic activity similar to that of piracetam, but not as strong [19]. Nootropic activity has also been reported in humans [2], but it was not specified whether these were healthy adult humans, and they were probably elderly or psychiatric patients.
Another trait phenibut shares with nootropics is neuroprotection. Multiple animal studies have indicated that phenibut administration increases resistance to the detrimental effects of edema on mitochondria and energy production in the brain [20-22]. Phenibut also normalizes brain energy metabolism changes caused by chronic stress [23]. It was found to prevent changes in plasma electrolytes caused by cerebral injury [24]. Phenibut also protects dopaminergic neurons, and improved the condition of patients being treated with antiparkinsonic drugs [25].
Other effects. Phenibut has anticonvulsant activity against some drugs or conditions, but not others. It also potentiates the action of some other anticonvulsant drugs, and has been used to treat patients with epilepsy [2]. Phenibut has been reported to reduce motion sickness, and used in the treatment of alcohol and morphine withdrawal [2, 26]. One study indicated that phenibut increased resistance to heat stress and improved working capacity in humans [27].
Some studies indicate that phenibut has anti-arrhythmic properties in humans [28-29]. It also has other cardioprotective properties [30-31]. Finally, phenibut showed promise in experimental models of gastric lesions [32-33].
Side effects and suggested use
Phenibut has low acute toxicity. Reported LD50s (dose required to kill 50% of laboratory animals) are 900 mg/kg i.p. in mice, 700 mg/kg i.p. in rats, and 1000 mg/kg in rats (method of administration not given) [2, 34]. Chronic administration of 50 mg/kg did not have teratogenic effects in rats [34]. In clinical studies, no signs of toxicity have been reported, and side effects are few. Some report drowsiness, but this effect is not nearly as likely or severe as with benzodiazepines [2].
One should be aware of the potential for drug interactions when taking phenibut. In many cases, it will decrease the threshold dose and potentiate certain actions of a drug. It amplifies some of the effects of anesthetics (ether, chloral hydrate, and barbiturates), diazepam, alcohol, and morphine [2, 35-36]; it would also presumably have an interaction with related drugs, such as other opiates and GHB. In contrast, taking phenibut with some other drugs, such as stimulants, will more than likely just blunt their effect.
In humans, the plasma half-life after a 250 mg oral dose of phenibut is 5.3 hours, and most of the administered drug is excreted unchanged [2]. Reported dosages used in clinical studies range from 250 to 1500 mg daily, usually divided among three doses [2, 37]. Feedback indicates that the ideal dose may be in the higher end of this range.
Tolerance develops to many of the effects of phenibut, although it is reported that it does not develop to the nootropic effect. The first signs of tolerance may be seen within as little as five days. For this reason, it is commonly used for one to two week periods, or dosage is increased by 25-30% after two weeks [2]. This makes phenibut ideal for short periods of stress or anxiety, but not ideal for chronic use. It is possible that taking only one dose daily may partially reduce the development of tolerance
phenbuit is something really cool it gives you a mild euphoric effect allmost like a high and the effects last for a good 18-24 hours. check out a review for one of the customer on my supp site i go to who used it:
I'm an idiot so I like to take things to the extreme. When I got my first 30g I took the 1.2g at about 6pm and went on about my night. I didn't feel anything too much so I decided to massive dose and took an additional ~4g (i figured this would be equiv to 8 gabatropins, the recommended high end massive dose, at 500mg each) at about 8pm. The next 2 hours I had friends over and we all were sipping beer and noticed the effects were much more pronounced. Kind of very drunk without the intoxication. When I went to bed at like 2am I was really "drunk" even though I had only 2 beers. My eyes kept crossing and 'wiggling' (like a strong dose of e does, not pleasant). I stumbled around and had to hold on to the walls to get to bed. When I closed my eyes I seemed to visualize highly creative patterns, it was similar to a milder version of lsd in this respect. When I woke up the next day I was ****ed up by the same amount and had to call into work sick because I had no concentration and was acting drunk, stumbling and slurring my words. The rest of the day i hung around the house and by evening I was starting to become un-drunk, but still had major effects in terms of highly increased socialability and lack of anxiety. I had a full hour conversation with my barber who I normally say <5 words to. Also insecurities that would normally be bothering me in the back of my head were non-existent. I rated this as neutral because want to people to read this post and understand that the effects of a ridiculously high dose (5g+) can be debilitating for a day or more. I was taken off guard at how strong and long lasting the effects were.
Since this I've used it off and on, not taking more that 2g in one day. Used properly this is the most effective supplement I've ever taken in regards to anxiety, expecially the social variety.
I'm an idiot so I like to take things to the extreme. When I got my first 30g I took the 1.2g at about 6pm and went on about my night. I didn't feel anything too much so I decided to massive dose and took an additional ~4g (i figured this would be equiv to 8 gabatropins, the recommended high end massive dose, at 500mg each) at about 8pm. The next 2 hours I had friends over and we all were sipping beer and noticed the effects were much more pronounced. Kind of very drunk without the intoxication. When I went to bed at like 2am I was really "drunk" even though I had only 2 beers. My eyes kept crossing and 'wiggling' (like a strong dose of e does, not pleasant). I stumbled around and had to hold on to the walls to get to bed. When I closed my eyes I seemed to visualize highly creative patterns, it was similar to a milder version of lsd in this respect. When I woke up the next day I was ****ed up by the same amount and had to call into work sick because I had no concentration and was acting drunk, stumbling and slurring my words. The rest of the day i hung around the house and by evening I was starting to become un-drunk, but still had major effects in terms of highly increased socialability and lack of anxiety. I had a full hour conversation with my barber who I normally say <5 words to. Also insecurities that would normally be bothering me in the back of my head were non-existent. I rated this as neutral because want to people to read this post and understand that the effects of a ridiculously high dose (5g+) can be debilitating for a day or more. I was taken off guard at how strong and long lasting the effects were.
Since this I've used it off and on, not taking more that 2g in one day. Used properly this is the most effective supplement I've ever taken in regards to anxiety, expecially the social variety.
